Research in the laboratory focuses on the understanding of the neurobiology of stress-related disorders, particularly Post-traumatic stress disorder and stress-induced depression, and the effects of stressful experiences in early life on cognitive and emotional abilities in adulthood.

Our team consists of graduate students, post-docs and reseacrch associates, both Israeli and international students, each with his or her own approach to studying stress-related dosorders. Aside from developing novel behavioral models, our lab uses a wide range of methods to study the neurobiological mechanisms underlying the behaviors. These include in vivo electrophysiology, microscopy and various molecular and biochemical analyses.

Our work involves both basic and clinical research and works in collaboration with the Institue for the Study of Affective Neuroscience (ISAN) in the University of Haifa.

For more information on our current projects visit our Research Interests section.

Recent Publications

Dorsal periaqueductal gray-amygdala pathway conveys both innate and learned fear responses in rats.

Kim EJ, Horovitz O, Pellman BA, Tan LM, Li Q, Richter-Levin G, Kim JJ. Proc Natl Acad Sci U S A. 2013 Sep 3;110(36):14795-800

In recent work done in collaboration with the Jeansok J. Kim lab of the University of Washington, we investigated the functional relationship between the  dorsal periaqueductal gray and amygdala. 

Read the paper


Prof. Gal Richter-Levin

gal fixedHead of Lab

Prof. Gal Richter-Levin is currently the head of the Institute for the Study of Affective Neuroscience (ISAN), at the University of Haifa.

He earned his PhD (1992) at the Weizmann Institute, Rehovot, Israel, under the supervision of Prof. Menahem Segal.

Prof. Richter-Levin joined the University of Haifa in 1995 and since 2006 he is a full professor at the Department of Neurobiology and the Department of Psychology.

Prof. Richter-Levin has made major contributions to developing novel translational animal models of mood and anxiety disorders, as well as towards the understanding of the role of emotional and amygdala activation in traumatic memory and depression.